The presence of poor attention in long COVID sheds light on the cognitive complexities of post-acute sequelae, emphasizing the need for tailored cognitive support and a nuanced approach to address the diverse challenges faced by individuals on the path to recovery.
Cognitive slowing emerges as a distinct marker in post-COVID-19 conditions, setting apart survivors with enduring symptoms. The pronounced delays observed in simple reaction time tasks signal a potential key factor contributing to cognitive impairments, marking a critical aspect of the post-COVID-19 cognitive landscape.
Mitochondria are often referred to as the powerhouse of cells, responsible for producing energy in the form of adenosine triphosphate (ATP) through oxidative phosphorylation. In Long COVID, mitochondrial dysfunction can occur due to various factors, including viral impact, immune system dysregulation, and oxidative stress, leading to impaired energy production and cellular processes.